Publications
Scientific Publications
Viva In Vitro receives the Impact Business 2023 recognition as a social and environmental impact initiative.
The Ship2B Foundation has communicated to the biomedical company Viva In Vitro on this February 15.— “After going through
NLRP3 and pyroptosis blockers for treating inflammatory diseases
Rebecca C. Coll, Kate Schroder, Pablo Pelegrín Highlights The nucleotide-binding oligomerization domain, leucine-rich repeat and pyrin domain-containing protein 3 (NLRP3)
Lipid-protein interactions regulating the canonical and the non-canonical NLRP3 inflammasome
Malvina Pizzuto, Pablo Pelegrin, Jean-Marie Ruysschaert Abstract The inflammatory response is a complex regulated effector mechanism of the innate immune
Galvanic current activates the NLRP3 inflammasome to promote Type I collagen production in tendon
Alejandro Peñin-Franch, José Antonio García-Vidal, Carlos Manuel Martínez, Pilar Escolar-Reina, Rosa M Martínez-Ojeda, Ana I Gómez, Juan M Bueno, Francisco
¿Se podría considerar la diabetes una enfermedad #inflamatoria? Nos preguntan los fundadores de Viva In Vitro en The Conversation España.
Creative commons, artículo original en: ¿Se podría considerar la diabetes una enfermedad inflamatoria? (theconversation.com). La inflamación es una respuesta coordinada
Hallan un nuevo inductor del proceso inflamatorio en enfermedades raras, cardiovasculares, degenerativas y metabólicas
Artículo original en Hallan un nuevo inductor del proceso inflamatorio en enfermedades raras, cardiovasculares, degenerativas y metabólicas | La Verdad
Sensing low intracellular potassium by NLRP3 results in a stable open structure that promotes inflammasome activation
Original document in Sensing low intracellular potassium by NLRP3 results in a stable open structure that promotes inflammasome activation (science.org)
P2X7 receptor induces mitochondrial failure in monocytes and compromises NLRP3 inflammasome activation during sepsis
Creative Commons, original document in P2X7 receptor induces mitochondrial failure in monocytes and compromises NLRP3 inflammasome activation during sepsis |